What is CCSVI?

The close spatial relationship between demyelization plaques and venous vessels on which CCSVI based, was postulated before 1980s as also the link between venous outflows disorders in the brain and the development back marks of MS.

“Already the next day I felt more strength, energy and walked more upright with a lighter step…” Peteriina Vainola-Grund

Multiple sclerosis (MS) has been considered for many years to be an autoimmune disease, but recent research from Italy points out a possible new cause. According to the theory of Prof. Paolo Zamboni, a vascular surgeon from Ferrara, narrowings (stenoses) in jugular veins (neck) or the azygos veins returning the blood from the head to the heart, might be responsible for the development and the symptoms of MS (Multiple sclerosis).

The blood vessels constriction prevents blood flowing quickly enough (so-called reflux, where the blood vessels does not work properly and the blood flows into the brain again), with the consequence that iron is deposited in the brain (it could be seemed as white plaques visible on the MRI scan) causing autoimmune inflammation damaging the protective myelin nerve coating in the spinal cord and in the brain.

This condition has become known as "CCSVI”: chronic cerebro-spinal venous insuffency.

Widening of the stenotic (narrowed) veins increases the blood flow through them, thus, relieving the back-pressure and eliminating the inflammation, which is causing the damage to the brain, the spinal cord and protective myelin.

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